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The therapeutic efficacy of adenoviral vectors for cancer gene therapy is limited by a low level of primary adenovirus receptors on tumour cells

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NK-EVs exert antitumor effects by enhancing the activation of specific apoptotic markers in hepatocellular carcinoma cells (HCC) (Hep3B) through the inhibition of serine/threonine protein kinase phosphorylation (Fig

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Quercetin attenuates chronic ethanol hepatotoxicity: Implication of free iron uptake and release
