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These oxidation reactions are triggered by environmental compounds that can cause cellular damage, potentially leading to health concerns

Parkinsons Disease (PD) involves degeneration of dopamine-producing neurons, mitochondrial dysfunction, alpha-synuclein aggregation, neuroinflammation, and gut-brain axis disturbances

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This suggests that cell wall adsorption alone is insufficient for meaningful AFB1 reduction in this strain and highlights the importance of enzymatic or transporter-mediated mechanisms that require live, metabolically active cells

Individuals with impaired methylation due to MTHFR, MTRR, or COMT gene mutations