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Close examination revealed that Sod2 +/ mutants display higher levels of oxidative damage, susceptibility to oxidative stress, inhibition of mitochondrial function, and premature initiation of apoptosis, yet exhibit close-to-normal lifespans (Li et al., 1995
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In addition, the shRNA-mediated downregulation of ATG12 resulted in the recurrence of efficacy for trastuzumab, erlotinib, gefitinib, and lapatinib in vitro (213)
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