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A trade-off switch of two immunological memories in Caenorhabditis elegans reinfected by bacterial pathogens

Accordingly, relying on our results, it can be hypothesized that direct glutathione infusion, through both activation of endogenous antioxidant defense mechanisms and attenuation of inflammatory cell recruitment, is able to protect vital myocardial components and endothelial cell function against a harmful prooxidant and inflammatory environment, thus preventing maladaptive cardiac repair and LV adverse remodeling

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These so-called reductive macrophages predominated and were associated with sustained maintenance of Th1 prevalence during aging until 2 years in the transgenic mice, while wild type littermates showed a rapid polarization to Th2 at 8 months