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Description

This anti-inflammatory action decreases oxidative stress across multiple organ systems, supporting its use in experimental protocols for chronic inflammation and metabolic disorders

The shift in their TH-1 or TH-2 systems remains constant and does not change over the course of the autoimmunity

Microglia play a crucial role in orchestrating this phenotypic shift: upon activation, they release inflammatory mediators such as IL-1, TNF- and C1q, which induce a pro-inflammatory reactive astrocyte phenotype (often termed A1-like or neurotoxic reactive state) (Zhao et al., 2024

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Results The sensory function in all nerve boundaries (peroneal, tibial, sural) returned to nearly normal by 8 weeks (Grade 2.7 on a scale of Grade 03 [0 = No function