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METH stimulates the release of excess glutamate from neurons while inhibiting its uptake by astrocytes, leading to the overactivation of NMDARs ( tThe excitotoxicity induced by METH also triggers neuroinflammation and amplifies its neurotoxic effects

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For wound healing or hair growth, 2 to 3 mg per day is common

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Global assessment of bisphenol A in the environment

Lancet 2001;358:17549.ArticlePubMed 33