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Studies have demonstrated that in pancreatic cancer, targeting Gln metabolism in conjunction with ERK signaling pathway inhibitors can halt pancreatic cancer cell metabolism and treatment resistance, thereby enhancing treatment outcomes (24)

Cell lines Human endothelial cells HUVEC (CC-Y1285) were purchased from EK-Bioscience (Shanghai, China)

This observation showed that it was possible to load the same bone twice in a different direction without changing its structure

This beneficial impact is achieved by directly inhibiting the formation of reactive oxygen species (ROS), which is mediated through fatty acid oxidation and a TGF- signaling pathway (Tanaka et al., 2008)

Other mechanisms possibly involved are glutathione and heme oxygenase-1 (174) as well as histone deacetylases expression (175)

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