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In an attempt to increase the NADH/NAD+ ratio and rescue cell viability, the MAS was inhibited by treatment with aminooxyacetic acid (AOA) that broadly inhibits transaminases including the aspartate aminotransferase of the MAS ( Fig

TMAO and the inflammasome NLRP3 The activation of the NLRP3 inflammasome is a critical mediator of TMAO-induced pro-inflammatory effects

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