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Together, these findings support a model in which OCTN1 acts as a context-sensitive regulator of the inflammatory-oxidative microenvironment, reflecting a dynamic balance between protective adaptation and pathological impairment, and providing a mechanistic view for its bidirectional regulation and potential as a therapeutic target

Salidroside pretreatment alleviates ferroptosis induced by myocardial ischemia/reperfusion through mitochondrial superoxide-dependent AMPKalpha2 activation

Autoantibodies and neurodegeneration in multiple sclerosis

Gut microbiota depletion by chronic antibiotic treatment alters the sleep/wake architecture and sleep EEG power spectra in mice

Lancet 365 , 15521560 (2005)