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Mitochondrial respiration reduces exposure of the nucleus to oxygen

doi: 10.1007/s11011-016-9948-1

In addition, this material proved itself convincingly in a retrospective study of hundreds of restorations of primary teeth

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Functional validation using receptor-deficient models confirms that 3-adrenergic signaling is required for these effects, as disruption of this pathway attenuates peptide-induced lipolysis

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