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This structural modification holds significant promise for targeted anti-fibrotic therapeutics

Cancer patients have an elevated level of DNA in their blood, which is caused by active release ( e.g

Harris et al., 2003

126 In vitro and in vivo experiments have demonstrated the role of NOX2-derived ROS as major redox signaling pathways in mediating the microglial response to stimulation with A42, the predominant A species found in amyloid plaques of AD patients

J Biol Chem 1955;217(1): 409-427
