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The possible mechanisms of arsenic-induced cardiotoxicity mainly include oxidative stress, DNA fragmentation, changes in cardiac ion channels (Figure 2, the possible arsenic exposure routes and mechanism of natural products against cadmium-induced cardiotoxicity have been summarized

Battaglia G, et al

10.1016/j.tem.2021.05.003 49 Hernndez-CuervoH.SoundararajanR.Sidramagowda PatilS.BreitzigM.AlleynM.GalamL.et al (2022)

In a similar way to that reported for sodium selenite, Kim and collaborators [71] demonstrated that superoxide anion generated by selenite triggered mitochondrial damage, subsequent mitophagy and cell death in glioma cells

All multilevel models were estimated on the basis of all the available data

Source: Koyuncu et al., 2025 Angiogenesis and Vascular Repair BPC-157 promotes VEGF (vascular endothelial growth factor) expression and the formation of new blood vessels