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In conclusion, OA pathogenesis is propelled by a metabolically dysregulated milieu, where obesity, oxidative stress, gut dysbiosis, and broader metabolic syndrome converge to create a self-perpetuating cycle of inflammation and tissue damage (Figure 1)

In addition, mTORC1 activation via glutaminolysis suppresses autophagy and the DNA damage response 164,165

Prioritizing restorative sleep, engaging in cognitive learning activities, and maintaining healthy stress management practices may further help reinforce neural resilience pathways associated with BDNF and synaptic plasticity

This stress can cause a decrease in the production of inflammatory cytokines like TNF- and interleukins (such as IL-6, IL-10, and IL-1), ultimately leading to the death of these cells

Abstract Heme is an iron-containing porphyrin compound widely used in the fields of healthcare, food, and medicine

MDA concentration = absorbance/1.56 105