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Description
A kinetic study of 1-anthraquinonesulfenic acid oxidation indicated a pH independent rate constant (85 M 1 s 1 )
Based on another study using kainate-induced rat models, PTGS2 expression was elevated in the CA3, cortex, and dentate gyrus regions 1824 h after seizures
Differential gene expression analysis was performed using limma 140
This high expression of GSTs mediates cellular resistance to antitumor drugs via distinct mechanisms, which mainly involve metabolic detoxification, regulation of the MAPK signaling pathway, DNA repair, autophagy, and glycolytic processes
10.1051/apido/2010039 459
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