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This polymorphism is in intron 13 of the MAO-B gene and is thought to disrupt monoamine metabolism, leading to increased ROS production and oxidative stress within the central nervous system (Leko et al., 2021)

AhR pathway ROS, especially H 2 O 2 , activate the AhR pathway by accelerating the production of endogenous ligands like FICZ and kynurenine (Kyn)

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Calcium supplementation also promotes calcium influx, activating the PKC pathway

For example, Nrf2 knockout in the aging mice reduced iron ions deposition in the brain, lowered the level of oxidative damage in the striatum, and also alleviated the age-related motor dysfunction [80]