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Protein oxidation leads to structural modifications, loss of function, and aggregation of pathological proteins, such as beta-amyloid in AD and alpha-synuclein in PD, which disrupt intracellular processes and amplify neurotoxicity [79,80,81]

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The molar excess of BCPB in relation to N-Hcy-Lys was 50:1

doi: 10.1016/j.cvfa.2020.03.008

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