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52,53 Therefore, it is possible that oral ALCAR/ ALA, as administered in this study, did not result in sufficient brain concentrations to significantly impact cerebral mitochondrial functioning

Mutation analysis in mitochondrial fatty acid oxidation defects: exemplified by acylCoA dehydrogenase deficiencies, with special focus on genotypephenotype relationship

Without an initial fasting period, on the first day of each new KD, patients received meals amounting to one-third of the planned daily energy requirement

Utter MF, Scrutton MC

The production of the toxin is tightly regulated it is mainly repressed by the presence of glucose in the vaginal tract [86]

Phelan K, McDermid HE