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doi: 10.4049/jimmunol.1900027

Subjects with pre-existing hypertension (defined as baseline systolic BP 130 mmHg) showed significantly higher adverse event rates: 32% experienced blood pressure increases exceeding 20 mmHg, compared to 6% in normotensive controls

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They exhibit a multifaceted mechanism of action, addressing oxidative stress, neurotransmitter imbalances, and neuroinflammation through Nf-B inhibition, replenishing GSH reserves, and alleviating the impairment of mGluR2 and mGluR3 [136, 205], acting on inflammatory state, lowering serum inflammatory cytokines and markers (IL-6, TNF-alfa, PCR) [161]