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In this Review, we describe the evidence for abnormalities in energy metabolism and mitochondrial function in migraine, with a focus on clinical data (including neuroimaging, biochemical, genetic and therapeutic studies), and consider the relationship of these abnormalities with the abnormal sensory processing and cerebral hyper-responsivity observed in migraine

Less ATP promotes more oxidative stress, meaning cells won't have the energy they need to function optimally

10.3390/ijms242316684 130 YongZ.RuiqiW.HongjiY.NingM.ChenzuoJ.YuZ.et al (2021)

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