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The mechanism of action of DSePA against RP involved its ability to suppress the radiation-induced sustained inflammatory response by upregulating the pulmonary activity level of GPx and concurrently blocking the NF-kB/IL-17/G-CSF/neutrophil axis (Table 2) (Fig

doi: 10.3390/polym12102403 37 MallmannMPOliveiraMS

In biochemical and cellular research, GHK-Cu is widely studied for its role in peptidemetal interactions, extracellular matrix signaling, copper-dependent molecular regulation, and cellular communication pathways