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but some of the soldiers receiving the drug became jaundiced and anaemic

Myelopathy results from the damage of nerve cells in a part of the spinal cord
(2008) described the discovery and characterization of the tissue-protective EPO receptor (now called the innate repair receptor) and demonstrated that engineered EPO derivatives lacking erythropoietic activity retained cytoprotective signaling, laying the foundation for ARA-290 development [2]

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