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While PKM2-targeted drug development predominantly centers on cancer metabolic modulation, preliminary investigations have begun exploring its therapeutic potential in neurodegenerative and inflammatory diseases

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In line with previous reports, we compared serum MOTS-c levels in human subjects with and without T1DM

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Although restricted to animal studies, the KD is related to several pathways in metabolic syndrome and cancer, including increased 5 adenosine monophosphate-activated protein kinase (AMPK) activity, inhibition of the mTOR/AKT pathway, 134 lowering the serum ratio of insulin-like growth factor/IGF-binding protein 3 135 and increasing peroxisome proliferator-activated receptor- coactivator-1 expression (a master mitochondrial metabolic regulator that can increase mitochondrial biogenesis)