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Iron accumulation in Alzheimer disease is a source of redox-generated free radicals
doi: 10.1007/s11064-021-03252-x
The pathogenicity of L
found that V-9302 inhibited the cell viability in a dose-dependent manner and promoted the apoptosis of AML cell lines HL-60 and KG-1 [52]
Control (n = 6), 5 mg/kg morphine (n = 6) and 3 BPC-157 groups (n = 18 in each group) and were examined using IHC
