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Importantly, we found that in 4T1 cells with moderate GSH level, it induced ferroptosis by consuming intracellular GSH and in turn inactivating GPX4

In the pathological processes of ferroptosis and renal fibrosis, crosstalk among ferroptosis, apoptosis, necroptosis, and autophagy plays a crucial role, with significant molecular and regulatory overlap

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10.3389/fpls.2017.01492 Front