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Cancer drug-tolerant persister cells: from biological questions to clinical opportunities

1c,d) and was previously suggested as a candidate for a potential activator site, first, because its positive charge might stabilize or bind GSH as GS , and, second, because of its evolutionary conserved position that is equivalent to the GSH-interacting catalytic residue of glutathione transferases 3

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(2012) reported that the disturbance in methionine metabolism, through SAM deficiency and consequently DNA hypomethylation impairs the morula-to-blastocyst transition during bovine preimplantation

The decreased activity of enzymes due to mercury intoxication is reflected by increased ROS-induced oxidative stress and the disruption of redox signaling pathways, which negatively affects neurodevelopment (Branco et al