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The R -gene mediated, hypersensitive-type resistance (HR, incompatible interaction) is based on a specific interaction, either directly or indirectly, of a bacterial effector gene product with the R gene of the host plant
Activators of FANCD2 have the potential to enhance its capacity to repair damaged DNA and shield cells from the detrimental consequences of DNA damage
Figure 3 We further investigated the role of aberrant DNA methylation in driving the pathogenic mechanisms of oxidative stress (Kowluru et al., 2016
G6PDH cryo-EM structures

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