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Polydopamine-based nanocomposite as a biomimetic antioxidant with a variety of enzymatic activities for Parkinsons disease

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The molecular mechanism underlying the KD-induced improvement of T2DM clinical outcomes has been investigated in both the system biology approach and mouse model studies

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Th2 cells, in SLE, promote disease initiation by causing autoantibody formation which lead to immune complex deposition and chronic inflammation induction

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