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Endogenous MOTS-c levels decline with age and correlate inversely with metabolic dysfunction

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1988), as well as with massive necrosis and an intense inflammatory reaction (Danan et al

PGC1-alpha over-expression prevents metabolic alterations and soleus muscle atrophy in hindlimb unloaded mice

By crossing the blood-brain barrier, it participates in supporting the synthesis of neurotransmitters involved in memory and attention, supplying energy to neurons and maintaining the normal functioning of neural networks

Oberst, A RIPK1 both positively and negatively regulates RIPK3 oligomerization and necroptosis Cell Death Differ 21 10: 1511-21