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Of these three mutants, YAP1 has previously been linked to carnitine-dependent phenotypes ( OPI3 and CHO2 encode enzymes that catalyze the final two sequential reactions in the pathway to synthesize phosphatidylcholine from phosphatidylethanolamine, and their co-selection from a screen of a genome-wide deletion strain library which only yielded three mutants strongly suggests a specific connection between these genes and carnitine-related activities

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In addition, GSK 3 has been demonstrated to be a transcription factor regulating tumor progression, invasion and metastasis (14,15)

Regulation of p53 and its downstream genes (e.g., SLC7A11, iPLA2, ALOX15, GLS2, TIGAR, PTGS2, FDR, DPP4, p21, and PARK2) may promote or inhibit the induction of ferroptosis

Almost all AKRs share a common protein structure, comprised of an (/)8-barrel with alternating eight -helices and eight -strands and a pyridine nucleotide binding site [39]