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dePadua, M

These active forms do not require conversion by the liver, which is often burdened by chronic inflammation and oxidative stress

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The specificity of G6PD induced upregulation was attributed to increased histone hyperacetylation by histone acetyltransferases (HAT)s and increased HDAC, HAT, DNA polymerase II, and SP1 (transcription factor) binding to the G6PD core promoter (Figure 3)

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