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However, the inhibition of VLDL uptake in mice overexpressing human apoC1 was also observed in the context of LDL-R deficiency, suggesting that apoC1 inhibition of VLDL uptake involves another pathway than LDL-R, probably LRP [65]

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If there is any doubt as to the aetiology of the lesion, referral to a relevant specialist is required (e.g

In part, it has to do with the drowsy hormone, melatonin

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Xu et al., 2020)