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while mitochondrial support may be genuinely life-changing for specific subsets of patients with measurable metabolic deficits, it is not a universal cure-all, emphasizing the urgent need for highly personalized medical approaches

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Several varieties of kinase proteins are generated during cellular stress, such as ERK1/2, MAP Kinase, and p38 MAP Kinase, as well as c-Jun N-terminal kinases (JNK) [60]