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Histological examination revealed visible collagenization and neovascularization causing closure of the tissue defect and alleviation of inflammation under the influence of BPC-157

Autophagy promotes ferroptosis by degradation of ferritin

Autry, A

[DOI] [PubMed] [Google Scholar] 16.Nguyen T, Nioi P, Pickett CB

From reports of influenza virus drug resistance mutations, we summarized that the mechanism of influenza virus NAI resistance mainly includes three mechanisms: first, catalytic amino acid mutation of the binding pocket causing a direct inhibitory effect, followed by framing or other amino acid site mutation causing an indirect structural hindrance, and finally amino acid site mutations at the monomer junction causing changes in monomer stability