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CIH, a core pathophysiological feature of OSA, acts as a key trigger for TGF- signaling and lung cancer progression, via two core regulatory mechanisms: first, CIH directly activates HIF-1, subsequently upregulating TGF- expression and driving Smad phosphorylation to induce myofibroblast differentiation and extracellular matrix (ECM) production (Liang et al., 2024)

Edaravone Acute Infarction Study, G., Effect of a novel free radical scavenger, edaravone (MCI-186), on acute brain infarction

Chem Rev 102(12):46394750 Vicik R, Busemann M, Baumann K, Schirmeister T (2006) Inhibitors of cysteine proteases

NAC may be a third way to restore disrupted mitophagy

26 Ferroptosis, an iron-dependent form of programmed cell death, has been implicated in endometrial damage

DB and SC acknowledge fellowship support from Council for Scientific and Industrial Research