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Since the consumption of NADPH by GSR may compete with NADPH-dependent inflammatory processes, we endeavored to determine NADPH consumption by GSR in macrophages (Ms) stimulated by lipopolysaccharide (LPS), a classical Toll-like receptor 4 ligand

This correlation between FASN activity and LD formation is likely important in supporting energy homeostasis and promoting tumour cell survival, especially under metabolic stress [119]

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Recently, emerging evidence has identified ferroptosis, an iron-dependent regulated cell death, as a pivotal factor in driving PD pathogenesis, with close associations to key mechanisms including -syn protein aggregation, excessive oxidative stress, mitochondrial dysfunction, disturbances in iron metabolism, and activation of neuroinflammatory responses