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Description
Their mechanisms of action are completely different, focusing on cellular repair and signaling pathways
RTW status was evaluated at 3 months
Conversely, BACH1 knockdown not only inhibited BPC157-induced HUVEC proliferation and tube formation but also blocked the expression of these proangiogenic factors, confirming that BACH1 is the pivotal downstream effector of the BPC157-FBXO22 axis
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Clevers HC, Bevins CL
December 5, 2025
