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In various pathological conditions implicating oxidative stress, ER functions are disturbed resulting in ER stress and initiation and propagation of unfolded protein response (UPR), which results in transient suppression of protein translation to allow recovery but leads to cell death when stress cannot be resolved (Soliman et al., 2019)

Summary Keywords allergic asthma, immunometabolism, innate immunity, innate lymphoid Cells (ILC2), pentose phosphate pathway, redox homeostasis and signaling, respiratory immunology Citation Cain J, Hurrell BP, Shen S, Speliakos P and Akbari O (2026) Melatonin suppresses ILC2-driven airway hyperreactivity via glutathione-dependent metabolic reprogramming

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Widely recognized in the research community for its high biological activity and stability, BPC-157 is a primary focus for Canadian studies involving accelerated tendon-to-bone healing, ligament repair, and the modulation of the nitric oxide (NO) system

BPC 157 was previously reviewed as novel mediator of Robert cytoprotection and endothelium protection in the stomach[8-11], and gut-brain axis[12], beneficial therapy in gastrointestinal tract[13,14], with particular reference to vascular recruitment[11,15-17], ulcerative colitis[14] and tumor cachexia[18], and other tissues healing[19,20]

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