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These findings highlight lipid peroxidation-related enzymes, particularly 12/15-LOX and ACSL4, as novel therapeutic targets for treating secondary brain injury post-stroke (Figure 2B)

Nutritional B vitamin deficiency disrupts lipid metabolism causing accumulation of proatherogenic lipoproteins in the aorta adventitia of ApoE null mice

PMID: 40683950 Neuron Oligodendrocyte-encoded lactate dehydrogenase A couples glycolysis to remyelination via protein lactylation

and R 1 is C 1-4 -alkylene-hydroxy

The underlying mechanisms of these complications are largely unidentified so far

doi: 10.1186/1472-6882-10-76 45 DostalAMArikawaAEspejoLBedellSKurzerMSStendell-HollisNR