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In this study, -tocotrienol was suggested to exert its effect by blocking the oligomerization of proapoptotic Bcl-2 proteins [167]
Cigarette smoke and CFTR: implications in the pathogenesis of COPD
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Mitochondrial stress has also been demonstrated to stimulate the release of molecules such as damage-associated molecular pattern (DAMP) proteins, which generate strong proinflammatory activity (Sliter et al., 2018)
