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Reyes JF, Ekmark-Lwen S, Perdiki M, Klingstedt T, Hoffmann A, Wiechec E, et al

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While IMCLs themselves can be benign energy stores, the specific accumulation of toxic lipid intermediates like DAGs and ceramides is central to pathology ( Concurrently, the liver avidly takes up the excess systemic FFAs, which serve as a substrate for the hepatic overproduction of triglyceride-rich VLDL ( Beyond this shared origin in lipid overflow, novel mechanisms underscore the direct crosstalk