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We have shown that H 2 S signaling is dysregulated in AD, and sulfhydration is diminished in the 3xTg-AD mouse model of AD as well as in human AD (Giovinazzo et al., 2 S produced by CSE sulfhydrates glycogen synthase kinase 3 (GSK3) and inhibits its catalytic activity (Figure 2)

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This area remains one of the most exciting frontiers for MOTS-c research, though it must be emphasized that these potential benefits are currently theoretical and extrapolated from its known biological functions and animal studies

2005;31 Spec No 2:5S205S26

The activation of CD36 can trigger multiple proinflammatory and profibrotic pathways, such as those involving Toll-like receptors, the NLRP3 inflammasome, PKC-NADPH oxidase, Src family kinases, MAPK, and TGF- [74]