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4 Discussion BPD is a severe chronic lung disease primarily impacting preterm infants, for which effective treatments remain lacking ( In vitro , Di-ART-GPC decreased inflammatory factor levels in hyperoxia-exposed macrophages while also elevating GPX4 expression, suggesting that Di-ART-GPC may protect against BPD lung injury by regulating macrophage ferroptosis

NADPH cytochrome P-450 reductase in rat, mouse and human brain

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Because ChaC1 is upregulated in DJ-1 knockout mice, DJ-1 is considered to maintain glutathione by suppressing ChaC1 expression, which thereby prevents PD development [115]

It is synthesized within the cells and plays a pivotal role in maintaining cellular redox balance, detoxification, and immune function