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Mechanistically, ferroptosis is distinct from other forms of programmed cell death: unlike apoptosis or pyroptosis, it does not depend on caspase activation, nor is it primarily driven by ATP depletion or mitochondrial ROS generation, which are central to necroptosis

Its presumed mechanism of action is through the glutamatergic system

Iron-sulphur cluster biogenesis and mitochondrial iron homeostasis

Neuroendoscopic parafascicular evacuation of spontaneous intracerebral hemorrhage (NESICH technique): a multicenter technical experience with preliminary findings