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Additionally, it has not been proven whether engineered bacteria might trigger an immune response

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This effect can be completely reversed by SOD2 expression, indicating that hyperglycemia-induced OXTR suppression is due to hyperglycemia-induced consistent oxidative stress, which has been termed hyperglycemia memory ( in vivo study, which showed that OXTR expression was suppressed in many brain tissues, including the amygdala, hypothalamus and hippocampus, in prenatal diabetes exposure-induced offspring

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The common cause is due to manganese deposition

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