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2023;8:eabq2424

3B), which are maintained by a set of enzymes, including apoplast-localized ascorbate oxidase and cytoplasmic dehydroascorbate reductases (Xiao et al

Biochemical analysis in 2,4-dienoyl-CoA reductase deficiency patients will show hyperlysinemia due to the requirement for NADPH of the mitochondrial lysine catabolic enzyme, -aminoadipate-6-semialdehyde synthase that is encoded by the AASS gene