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An example was presented by Grant et al., [162] with romidepsin that is activated by intracellular GSH by cleavage of a disulfide bond
Study results are conflicting
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Each sample was reconstituted with 270 L phosphate buffer in D 2 O
It has been well documented that sepsis occurs because of over-activated immune responses which include intractable inflammatory responses associated with imbalanced cytokine production (18)
