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10.1111/j.1755-5949.2012.00338.x 55 LiuS

It inhibits NOTCH1 signaling by using its GCT activity to cleave a -glutamyl-cysteine bond at glutamate 1669 of the immature NOTCH receptor, thereby preventing the essential S1 cleavage and subsequent activation ( FIGURE 6 The physiological importance of this mechanism is underscored by severe developmental phenotypes upon its disruption

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Indeed, 14-3-3 inhibition by SELENOW in breast cancer cells was found to increase cell proliferation and cell survival through increasing resistance to genotoxic stress in human breast and lung cancer cells (22)

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