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For instance, it remains unclear how ferroptosis in neutrophils [e.g., GPX4 deficiency-induced iron release (216)] directly modulates T/B-cell differentiation or macrophage activation, or whether DAMPs released by ferroptotic immune cells propagate ferroptosis in renal parenchymal cells (e.g., tubular epithelial cells)

Liu N, Wang LH, Guo LL, Wang GQ, Zhou XP, Jiang Y

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In patients, activation of the NRF2 pathway is associated with more aggressive disease (Homma et al, 2009

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