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In conclusion, our structural and functional analyses provide a large body of novel information for defining the molecular mechanism underlying the activation of c-MET in response to HGF and NK1 and paves the way for the eventual therapeutic intervention of diseases caused by aberrant activation of c-MET

In this context, the evaluation of the difference in tumor growth between humanized HLA mice and athymic nude mice can provide a clear view of the role of immune cells in the tumor suppressor role of mGluR1 in glioma

However, unlike in control cells, the activities of ACO2 and the Fe-S dependent respiratory supercomplexes were diminished to varying degrees following 20 h of cystine starvation in CHAC1-deficient cells (Supplementary Fig

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Following a thorough screening process, 358 research articles were selected for the review

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