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These studies suggest that EE interventions may promote endogenous neurogenesis, leading to long-term improvements in neurological deficits among children with HIBD

SIRT6 alleviates mitochondrial dysfunction, podocyte injury, and ferroptosis induced by HG through activating the Nrf2/GPX4 pathway and increasing SLC7A11, HO-1, and SOD2 expressions

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10.1007/s00018-022-04365-4 [DOI] [PMC free article] [PubMed] [Google Scholar] 313.Kim, K

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